Some people dont think what you eat and drink effects your health ! (or what think, do , activities...)
If someone drinks soda and find a healthy or at least healthier alternitive. If eat lots of processed sugar and foods, can try to find good substitute. People are dying and feel so little is said on radio, TV , newspapers on helping to stay healthier and avoid illnesses. Who is controling all those "news" outlets?
Try to find more REAL food, not so much fried foods (like potato chips...). Most are made with high omega 6 oils which we need, but not at expense of eating omega 3 fats like in some seeds but mostly fish. I am hoping you all are increasing in your health and have found ways to share info. Have also found somethings like acupressure /reflexology/Pain Erasure(Bonnie)/Trigger pt... which are all type of healing self and we all may do it by rubbing forehead for head pain .....
http://www.katolenyardley.com/circulation_choices.pdf Bioflavonoids are found in peppers, buckwheat, aprico
https://www.hormonesmatter.com/covid-notes-blood-clots-problem/
Really interesting on Thiamine deficiency being much more common than thought.
https://www.frontiersin.org/articles/10.3389/fpsyt.2019.00207/full Neurological, Psychiatric, and Biochemical Aspects of Thiamine Deficiency in Children and Adults Shibani Dhir1†, Maya Tarasenko1†, Eleonora Napoli1 and Cecilia Giulivi1,2* 1Department of Molecular Biosciences, School of Veterinary Medicine, University of California, Davis, Davis, CA, United States brain is highly vulnerable to thiamine deficiency due to its heavy reliance on mitochondrial ATP production. body does not store thiamine in levels >30 mg, and the half-life for thiamine is only 9–18 days. proportionally increase thiamine’s dietary demand (a minimum of 0.33 mg per 1,000 kcal) (1). Thus, rather than focusing on thiamine’s RDA, it is critical to match its intake with carbohydrate consumption as well as total caloric intake. thiamine inactivation by heat and/or sulfur dioxide deficiency can result from genetic factors, i.e., pathogenic gene mutations in key regulators of the thiamine pathway, including thiamine pyrophosphokinase 1 (TPK1), thiamine diphosphate kinase (TDPK), thiamine triphosphatase (THTPA), and thiamine transporters (SLC25A19, SLC19A2/THTR1, and SLC19A3/THTR2; Figure 1B). More recently, the organic cation transporter 1 (OCT1) has been claimed to act as a hepatic thiamine transporter (25).
https://www.cell.com/ajhg/fulltext/S0002-9297(11)00485-X
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